By editors, Stefan Mauss ... et al. .
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Extra resources for Hepatology a clinical textbook 2010 - 2nd edition
The risk of severe hepatitis and fulminant hepatic failure seems to be elevated if both infections occur simultaneously regardless of whether it is an acute coinfection of HBV and HCV or acute hepatitis C in chronic hepatitis B (Liaw 2004). Hepatitis D coinfection: Acute HBV and HDV coinfection tends to be more severe than acute HBV infection alone. It is more likely to result in fulminant hepatitis. If HDV superinfection in patients with chronic HBV infection occurs, HDV usually predominates, and HBV replication is suppressed (Jardi 2001).
For example, transforming growth factor B1 (TGF B1) phenotype and fibrosis stage are correlated. Patients with moderate to severe steatosis are at higher risk for developing hepatic fibrosis. Acute hepatitis B in a patient with chronic hepatitis C may be more severe. Chronic hepatitis B may be associated with decreased HCV replication as opposed to HCV monoinfected patients, al- 25 Hepatology - A clinical textbook though HCV usually predominates. , HBsAg) although HBV DNA is detectable. • Geography and environmental factors: There are some obvious geographic differences (Lim 2008).
Natural history of chronic hepatitis B: special emphasis on disease progression and prognostic factors. J Hepatol 2008;48(2):335. Fung J, Lai CL, Yuen MF. New paradigms for the treatment of chronic hepatitis B. J Gastroenterol Hepatol 2008;23(8 Pt 1):1182. Ganem D, Prince AM. Hepatitis B virus infection--natural history and clinical consequences. N Engl J Med 2004;350(11):1118. Garfein RS, Bower WA, Loney CM, et al. Factors associated with fulminant liver failure during an outbreak among injection drug users with acute hepatitis B.
Hepatology a clinical textbook 2010 - 2nd edition by editors, Stefan Mauss ... et al. .